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Finally, we summarized the present knowledge about the Trx/TrxR inhibitors as anticancer drugs

Tissue damage developed after MTX utilization is caused by ROS which mediates destruction of lipids resulting in a breakdown of cell membrane and disturbance of physiological processes (ener et al

The NO system modulation appears bidirectional, with BPC-157 enhancing beneficial NO signaling while potentially limiting excessive or damaging NO production in inflammatory conditions