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10.1021/bi960737u 59 MojzikovaR.KoralkovaP.HolubD.ZidovaZ.PospisilovaD.CermakJ.et al (2014)

[DOI] [PubMed] [Google Scholar] 317.Chen K.H., Hsiang E.L., Hsu M.Y., Chou Y.C., Lin T.C., Chang Y.L., et al

During this process, PLA particles were colonized by gut microbes, forming surface biofilms and increasing pullulanase activityan enzyme that degrades branched polysaccharidesthe authors suggested this may have broken down PLA into smaller carbon-rich molecules, potentially supporting Bifidobacterium growth, without triggering pathogenic overgrowth or inflammatory responses (Jimnez-Arroyo et al., 2023)

Subsequent research indicated that genistein reduces oxidative stress and myogenic apoptosis through various mechanisms, including the regulation of ROS, lipid peroxidation, Bcl-2, and the apoptosis marker caspase-3 via the PI3K-Akt and ERK1/2 MAPK signaling pathways, with its effects remaining unaffected by estrogen receptor antagonists [28]