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Illustratively, BPC 157 therapy attenuated/eliminated the whole BuddChiari syndrome [26] (i.e., left superior caval vein-azygos vein-inferior caval vein shunt to skip the suprahepatic occlusion of the inferior caval vein), including the counteraction of the prominent deadly syndrome

28 For the specific classification, function, and mechanism of AATs in the human body, we invite readers to review the following literature

Oxidative stress also activates the Kelch-like ECH-associated protein 1 (Keap1)-Nuclear factor erythroid 2-related factor 2 (Nrf2)-antioxidant response element (ARE) redox regulator pathway, releasing Nrf2 to regulate the expression of genes that control antioxidant, inflammatory and immune system responses, facilitating GSH activity